[Consumer wearables, cardiology and clinical practice].

A 29-year-old male with prior testicular cancer and daily ketamine use developed severe lower urinary tract symptoms due to bladder fibrosis. Despite multiple treatments - botulinum toxin type A , bladder distension, and medications - his condition progressed to hydronephrosis, recurrent urosepsis, and acute kidney injury requiring nephrostomies. Eventually, a Bricker bladder was created. The case highlights ketamine-associated uropathy as an ongoing and underrecognized issue, stressing the importance of early cessation and targeted intervention.